Research output per year
Research output per year
Personal profile
Biography
Holly Stefen was awarded a Bachelor of Psychological Science majoring in Neuroscience from UNSW, Sydney, in 2015. After completing her undergraduate Honours year under the supervision of Prof Thomas Fath in the Neurodegeneration and Repair Unit, Holly began working as a Research Assistant within the unit. In December 2018, the Neurodegeneration and Repair Unit moved to Macquarie University as part of the Dementia Research Centre (DRC), where Holly continues her work as a Research Officer.
Research interests
During her time within the Neurodegeneration and Repair Unit and the Dementia Research Centre, Holly Stefen has developed an interest in understanding the role of the neuronal cytoskeleton in healthy and diseased states. Her work aims to uncover the contribution of actin-associated proteins in neuronal morphology, function and regeneration. Her interests have led her to acquire various technical skills including primary neuron cell culture, fluorescence imaging techniques, and more recently, expansion microscopy.
Education/Academic qualification
Bachelor Psychological Science (Hon), Neuroscience, The University of New South Wales
Feb 2012 → Dec 2015
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Collaborations and top research areas from the last five years
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Research Outputs
- 18 Article
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Tau target identification reveals NSF-dependent effects on AMPA receptor trafficking and memory formation
Prikas, E., Paric, E., Asih, P. R., Stefanoska, K., Stefen, H., Fath, T., Poljak, A. & Ittner, A., 15 Sept 2022, In: EMBO Journal. 41, 18, p. 1-24 24 p., e10242.Research output: Contribution to journal › Article › peer-review
6 Citations (Scopus) -
Deletion of the actin-associated tropomyosin Tpm3 leads to reduced cell complexity in cultured hippocampal neurons: new insights into the role of the C-terminal region of Tpm3.1
Tomanic, T., Martin, C., Stefen, H., Paric, E., Gunning, P. & Fath, T., 23 Mar 2021, In: Cells. 10, 3, p. 1-21 21 p., 715.Research output: Contribution to journal › Article › peer-review
Open AccessFile7 Citations (Scopus)57 Downloads (Pure) -
Overexpression of tropomyosin isoform tpm3.1 does not alter synaptic function in hippocampal neurons
Chaichim, C., Tomanic, T., Stefen, H., Paric, E., Gamaroff, L., Suchowerska, A. K., Gunning, P. W., Ke, Y. D., Fath, T. & Power, J., 1 Sept 2021, In: International Journal of Molecular Sciences. 22, 17, p. 1-16 16 p., 9303.Research output: Contribution to journal › Article › peer-review
Open AccessFile50 Downloads (Pure) -
Syntaxins 6 and 8 facilitate tau into secretory pathways
Lee, W. S., Tan, D. C. S., Deng, Y., van Hummel, A., Ippati, S., Stevens, C., Carmona-Mora, P., Ariawan, D., Hou, L., Stefen, H., Tomanic, T., Bi, M., Tomasetig, F., Martin, A., Fath, T., Palmer, S., Ke, Y. D. & Ittner, L. M., 16 Apr 2021, In: Biochemical Journal. 478, 7, p. 1471–1484 14 p.Research output: Contribution to journal › Article › peer-review
Open AccessFile4 Citations (Scopus)15 Downloads (Pure) -
CYLD is a causative gene for frontotemporal dementia - amyotrophic lateral sclerosis
Dobson-Stone, C., Hallupp, M., Shahheydari, H., Ragagnin, A. M. G., Chatterton, Z., Carew-Jones, F., Shepherd, C. E., Stefen, H., Paric, E., Fath, T., Thompson, E. M., Blumbergs, P., Short, C. L., Field, C. D., Panegyres, P. K., Hecker, J., Nicholson, G., Shaw, A. D., Fullerton, J. M., Luty, A. A., & 19 others, 1 Mar 2020, In: Brain : a journal of neurology. 143, 3, p. 783-799 17 p.Research output: Contribution to journal › Article › peer-review
53 Citations (Scopus)