Disruption to intracellular trafficking as a central pathogenic mechanism in amyotrophic lateral sclerosis

Atkin, Julie (Primary Chief Investigator)
Cole, Nicholas (Chief Investigator)
Chung, Roger (Chief Investigator)
Rizos, Helen (Associate Investigator)
Bell, Toby (Associate Investigator)

Department of Biomedical Sciences

Project: Research

Status	Active
Effective start/end date	1/01/15 → …

Related content

Research Outputs

A novel amyotrophic lateral sclerosis mutation in OPTN induces ER stress and Golgi fragmentation in vitro

Research output: Contribution to journal › Article › Research › peer-review

Protein Disulphide Isomerases: emerging roles of PDI and ERp57 in the nervous system and as therapeutic targets for ALS

Research output: Contribution to journal › Review article › Research › peer-review

The unfolded protein response and the role of Protein Disulfide Isomerase in neurodegeneration

Research output: Contribution to journal › Article › Research › peer-review

Golgi fragmentation in amyotrophic lateral sclerosis, an overview of possible triggers and consequences

Research output: Contribution to journal › Review article › Research › peer-review