Exhaled nitric oxide is decreased by exposure to the hyperbaric oxygen therapy environment

Zudin A. Puthucheary; Jia Liu; Michael Bennett; Barbara Trytko; Sharron Chow; Paul S. Thomas

doi:10.1155/MI/2006/72620

Exhaled nitric oxide is decreased by exposure to the hyperbaric oxygen therapy environment

Zudin A. Puthucheary^*, Jia Liu, Michael Bennett, Barbara Trytko, Sharron Chow, Paul S. Thomas

^*Corresponding author for this work

Research output: Contribution to journal › Article › peer-review

23 Citations (Scopus)

Abstract

Exhaled nitric oxide (eNO) detects airway inflammation. Hyperbaric oxygen therapy (HBOT) is used for tissue hypoxia, but can cause lung damage. We measured eNO following inhalation of oxygen at different tensions and pressures. Methods. Part 1, eNO was measured before and after HBOT. Part 2, normal subjects breathed 40% oxygen. Results. Baseline eNO levels in patients prior to HBOT exposure were significantly higher than in normal subjects (P

Original language	English
Article number	72620
Pages (from-to)	1-6
Number of pages	6
Journal	Mediators of Inflammation
DOIs	https://doi.org/10.1155/MI/2006/72620
Publication status	Published - 2006
Externally published	Yes

Keywords

SYNTHASE GENE-EXPRESSION
LUNG EPITHELIAL-CELLS
RADICAL PRODUCTION
INTERFERON-GAMMA
INDUCED SHOCK
HYPEROXIA
CHILDREN
INJURY
RAT
RECOMMENDATIONS

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title = "Exhaled nitric oxide is decreased by exposure to the hyperbaric oxygen therapy environment",

abstract = "Exhaled nitric oxide (eNO) detects airway inflammation. Hyperbaric oxygen therapy (HBOT) is used for tissue hypoxia, but can cause lung damage. We measured eNO following inhalation of oxygen at different tensions and pressures. Methods. Part 1, eNO was measured before and after HBOT. Part 2, normal subjects breathed 40\% oxygen. Results. Baseline eNO levels in patients prior to HBOT exposure were significantly higher than in normal subjects (P",

keywords = "SYNTHASE GENE-EXPRESSION, LUNG EPITHELIAL-CELLS, RADICAL PRODUCTION, INTERFERON-GAMMA, INDUCED SHOCK, HYPEROXIA, CHILDREN, INJURY, RAT, RECOMMENDATIONS",

author = "Puthucheary, \{Zudin A.\} and Jia Liu and Michael Bennett and Barbara Trytko and Sharron Chow and Thomas, \{Paul S.\}",

year = "2006",

doi = "10.1155/MI/2006/72620",

language = "English",

pages = "1--6",

journal = "Mediators of Inflammation",

issn = "0962-9351",

publisher = "John Wiley \& Sons",

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T1 - Exhaled nitric oxide is decreased by exposure to the hyperbaric oxygen therapy environment

AU - Puthucheary, Zudin A.

AU - Liu, Jia

AU - Bennett, Michael

AU - Trytko, Barbara

AU - Chow, Sharron

AU - Thomas, Paul S.

PY - 2006

Y1 - 2006

N2 - Exhaled nitric oxide (eNO) detects airway inflammation. Hyperbaric oxygen therapy (HBOT) is used for tissue hypoxia, but can cause lung damage. We measured eNO following inhalation of oxygen at different tensions and pressures. Methods. Part 1, eNO was measured before and after HBOT. Part 2, normal subjects breathed 40% oxygen. Results. Baseline eNO levels in patients prior to HBOT exposure were significantly higher than in normal subjects (P

AB - Exhaled nitric oxide (eNO) detects airway inflammation. Hyperbaric oxygen therapy (HBOT) is used for tissue hypoxia, but can cause lung damage. We measured eNO following inhalation of oxygen at different tensions and pressures. Methods. Part 1, eNO was measured before and after HBOT. Part 2, normal subjects breathed 40% oxygen. Results. Baseline eNO levels in patients prior to HBOT exposure were significantly higher than in normal subjects (P

KW - SYNTHASE GENE-EXPRESSION

KW - LUNG EPITHELIAL-CELLS

KW - RADICAL PRODUCTION

KW - INTERFERON-GAMMA

KW - INDUCED SHOCK

KW - HYPEROXIA

KW - CHILDREN

KW - INJURY

KW - RAT

KW - RECOMMENDATIONS

UR - https://www.scopus.com/pages/publications/33749524986

U2 - 10.1155/MI/2006/72620

DO - 10.1155/MI/2006/72620

M3 - Article

C2 - 17392577

SN - 0962-9351

SP - 1

EP - 6

JO - Mediators of Inflammation

JF - Mediators of Inflammation

M1 - 72620

ER -

Exhaled nitric oxide is decreased by exposure to the hyperbaric oxygen therapy environment

Abstract

Keywords

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