Impact of β-amyloid on the tau pathology in tau transgenic mouse and tissue culture models

Jürgen Götz; Della C. David; Lars M. Ittner

doi:10.1007/978-1-84628-440-3_12

Impact of β-amyloid on the tau pathology in tau transgenic mouse and tissue culture models

Jürgen Götz, Della C. David, Lars M. Ittner

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

Abstract

Dementia is a generic term that describes chronic or progressive dysfunction of cortical and subcortical functions that result in complex cognitive decline. These cognitive changes are commonly accompanied by disturbances of mood, behavior, and personality. In developed countries with an increasingly aging population, the prevalence of dementia is currently at around 1.5% at 65 years of age, which doubles every 4 years and reaches about 30% at the age of 80.

Original language	English
Title of host publication	Abeta peptide and Alzheimer's disease
Subtitle of host publication	celebrating a century of research
Editors	Colin J. Barrow, David H. Small
Place of Publication	London
Publisher	Springer, Springer Nature
Chapter	12
Pages	198-215
Number of pages	18
ISBN (Electronic)	9781846284403
ISBN (Print)	1852339616, 9781852339616
DOIs	https://doi.org/10.1007/978-1-84628-440-3_12
Publication status	Published - 1 Dec 2007
Externally published	Yes

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10.1007/978-1-84628-440-3_12

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Impact of β-amyloid on the tau pathology in tau transgenic mouse and tissue culture models. / Götz, Jürgen; David, Della C.; Ittner, Lars M.
Abeta peptide and Alzheimer's disease: celebrating a century of research. ed. / Colin J. Barrow; David H. Small. London: Springer, Springer Nature, 2007. p. 198-215.

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

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Impact of β-amyloid on the tau pathology in tau transgenic mouse and tissue culture models

Abstract

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