Remyelination trials: are we expecting the unexpected?

Alexander Klistorner, Michael Barnett

    Research output: Contribution to journalReview articlepeer-review

    11 Citations (Scopus)
    65 Downloads (Pure)

    Abstract

    Neuroaxonal loss is believed to underpin the progressive disability that characterizes multiple sclerosis (MS). While focal inflammatory demyelination is a principal cause of acute axonal transection and subsequent axonal degeneration, the gradual attrition of permanently demyelinated axons may also contribute to tissue damage, particularly in the progressive phase of the disease. Therefore, remyelination is considered a putative neuroprotective strategy. In this article, we review the potential pitfalls of remyelination trials, provide a framework for their appropriate design and temper the expectations, at times unrealistic, of researchers, regulators and the pharmaceutical industry.

    Original languageEnglish
    Article numbere1066
    Pages (from-to)1-5
    Number of pages6
    JournalNeurology: Neuroimmunology and Neuroinflammation
    Volume8
    Issue number6
    DOIs
    Publication statusPublished - 1 Nov 2021

    Bibliographical note

    Copyright the Author(s) 2021. Version archived for private and non-commercial use with the permission of the author/s and according to publisher conditions. For further rights please contact the publisher.

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